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Thus, amygdalin has been shown in vitro experiments to induce apoptosis due to upregulated expression of caspase-3 and Bax protein and downregulated expression of Bcl-2, an anti-apoptotic protein (217) ( Table 2 )

During neurodegenerative processes, microglial activation is frequently observed, with increasing evidence suggesting that iron overload and disrupted iron homeostasis contribute to neurodegeneration in AD (47,48)

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17-hydroxyprogesterone levels may be necessary to rule out an adrenal cause of androgen excess